An thiol-dependent isopeptidase activity that cleaves ubiquitin from a target protein to which it is conjugated.

The chemical reactions and pathways resulting in the breakdown of nicotinate.

Catalysis of the cleavage of the N-C1' glycosidic bond between the damaged DNA base and the deoxyribose sugar, releasing a free base and leaving an apyrimidinic (AP) site. Enzymes with this activity recognize and remove uracil bases in DNA that result from the deamination of cytosine or the misincorporation of dUTP opposite an adenine.

The chemical reactions and pathways resulting in the formation of nicotinate.

Catalysis of the reaction: diphosphate + UMP = 5-phospho-alpha-D-ribose 1-diphosphate + uracil.

Catalysis of the reaction: urate + O2 + H2O = 5-hydroxyisourate + hydrogen peroxide.

Catalysis of an oxidation-reduction (redox) reaction in which a nitrogenous group, excluding NH and NH2 groups, acts as a hydrogen or electron donor and reduces oxygen.

Catalysis of the reaction: ATP + bicarbonate + urea = ADP + 2 H(+) + phosphate + urea-1-carboxylate.

Catalysis of the reaction: (S)-ureidoglycolate + H(2)O + 2 H(+) = CO(2) + glyoxylate + 2 NH(4)(+).

Catalysis of the reaction: ATP + uridine = ADP + UMP.

Any process that stops, prevents or reduces the frequency, rate or extent of natural killer cell chemotaxis.

Any process that modulates the frequency, rate or extent of natural killer cell chemotaxis.

Any process that activates or increases the frequency, rate or extent of natural killer cell chemotaxis.

OBSOLETE. Any process that activates or increases the frequency, rate or extent of induction of apoptosis in response to chemical stimulus.

Any process that activates or increases the frequency, rate or extent of blood vessel remodeling.

Any process that modulates the rate, frequency or extent of blood vessel remodeling, the reorganization or renovation of existing blood vessels.

The chemical reactions and pathways involving 7,8-didemethyl-8-hydroxy-5-deazariboflavin.