The series of molecular signals in which an intracellular signal is conveyed to trigger the apoptotic death of a cell. The pathway starts with reception of an intracellular signal (e.g. DNA damage, endoplasmic reticulum stress, oxidative stress etc.), and ends when the execution phase of apoptosis is triggered. The intrinsic apoptotic signaling pathway is crucially regulated by permeabilization of the mitochondrial outer membrane (MOMP).

A process that begins with venom being forced into an organism by the bite or sting of another organism, and ends with hemolysis in the bitten organism.

Any process that stops, prevents or reduces the frequency, rate or extent of estradiol secretion.

Any process that modulates the frequency, rate or extent of estradiol secretion.

A heterodimeric protein complex which catalyses sulfur transfer from the sulfur carrier subunit of MPT synthase to precursor Z to synthesize MPT as part of molybdopterin cofactor (Moco) biosynthesis. In E. coli the subunits are MoaE and MoaD; in human, MOCS2B and MOCS2A. Moco biosynthesis and its constituent molecules are evolutionarily conserved.

A protein complex capable of catalyzing the transfer of sulfur atoms from one compound (donor) to another (acceptor).

Any process that activates or increases the frequency, rate or extent of estradiol secretion.

Any process that activates or increases the frequency, rate or extent of steroid hormone secretion.

Any process that activates or increases the frequency, rate or extent of estrogen secretion.

Any process that activates or increases the frequency, rate or extent of estrone secretion.

Any process that modulates the frequency, rate or extent of estrone secretion.

Any process that stops, prevents or reduces the frequency, rate or extent of estrone secretion.

Any process that activates or increases the frequency, rate or extent of progesterone secretion.

Any process that activates or increases the frequency, rate or extent of female gonad development.

Any process that modulates the frequency, rate or extent of progesterone secretion.

Any process that modulates the frequency, rate or extent of histone H4 acetylation involved in response to DNA damage stimulus.

Any process that modulates the frequency, rate or extent of response to DNA damage stimulus.

Any process that modulates the rate, frequency, or extent of histone H4 acetylation, the modification of histone H4 by the addition of an acetyl group.

Any process that modulates the frequency, rate or extent of female gonad development.